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A) 1.0 B) 1.5 C) 2.0 D) 2.5
A) Two times B) Five times C) Three times D) Ten times
A) Eight times B) Two times C) Five times D) Ten times
A) High risk of stent thrombosis B) Risk of INR fluctuation C) Risk of internal hemorrhage D) Risk of vitamin K deficiency
A) Recombinant factor VIIa B) Vitamin K C) Prothrombin complex concentrates D) Fresh frozen plasma
A) High risk of infection B) Slow onset of action C) High cost of treatment D) High risk of thromboembolism
A) To improve INR stability B) To avoid liver damage C) To prevent allergic reactions D) To reduce the time for rewarfarinisation
A) Prothrombin complex concentrates B) Activated charcoal C) Fresh frozen plasma D) Vitamin K
A) 20% to 30% B) 10% to 20% C) 5% to 10% D) 1% to 3%
A) 327,000 B) 200,000 C) 400,000 D) 301,000
A) 30% B) 20% C) 40% D) 80%
A) 70% B) 50% C) 40% D) 20%
A) 20% B) 70% C) 40% D) 80%
A) Airborne droplets B) Mosquito bites C) Sharing food utensils D) Direct skin contact
A) Elimination of sugar from diets B) Widespread vaccination C) New effective acid suppressants D) Increased smoking cessation
A) Increased use of anticoagulants B) Rational use of NSAIDs C) Higher reliance on warfarin D) Increased intake of vitamin K
A) Easier to reverse with vitamin K B) Cheaper in cost C) Fewer interactions with food D) More effective at preventing thromboembolism
A) Gastric ulcer B) Esophageal ulcer C) Meckel's diverticulum ulcer D) Duodenal ulcer
A) Type II B) Type V C) Type III D) Type I
A) Irregular with elevated borders B) Smooth with no elevation C) Regular with elevated borders D) Perpendicular with no surrounding inflammation
A) Fibrin, collagen, epithelial cells, and inflammatory exudate B) Granuloma, fibrous tissue, necrotic debris, and mucus C) Fibrinoid necrosis, inflammatory exudate, granulation tissue, and fibrous tissue D) Epithelial tissue, muscle tissue, blood vessels, and nerves
A) H2 antagonist B) Proton pump inhibitor (PPI) C) Aspirin D) Misoprostol
A) It promotes abortion and causes gastrointestinal distress B) It interacts negatively with proton pump inhibitors C) It is ineffective against gastric ulcers D) It has high cardiovascular risks
A) Bismuth-containing quadruple therapy B) Immediate surgery C) Triple regimen therapy D) Increased dosage of amoxicillin
A) 80% B) 60% C) 90% D) 70%
A) Type IV B) Type I C) Type III D) Type II
A) Muscularis mucosae and lamina propria B) Submucosa and adventitia C) Serosa and muscularis propria D) Epithelium and basement membrane
A) Naproxen with PPI B) Aspirin with H2 antagonist C) Celecoxib with misoprostol D) Ibuprofen with PPI
A) It drops below 50% B) It decreases C) It increases D) It stays the same
A) Irregular B) Perpendicular C) Elevated and regular D) Smooth
A) Immediate healing B) Increased appetite C) Catastrophic consequences D) Chronic constipation
A) Viral gastroenteritis B) Acute chemical peritonitis C) Systemic hypoglycemia D) Pulmonary embolism
A) Blurred vision B) High fever C) Severe diarrhea D) Sudden intense abdominal pain
A) Valentino's syndrome B) Zollinger-Ellison syndrome C) Conn's syndrome D) Cushing's syndrome
A) 10% B) 50% C) 5% D) 20%
A) A narrowing of the pyloric canal B) The thickening of the stomach wall C) The spread of bacteria to the lungs D) A hole leading into adjacent organs
A) Hyperchlorhydria B) Stenosis C) Metaplasia D) Peritonitis
A) Severe vomiting B) Internal bleeding C) High fever D) Severe constipation
A) Stool culture B) Biopsies C) Blood tests D) Physical examination
A) Pepsin B) Amylase C) Lipase D) Urease
A) BabA B) Insulin C) Cortisol D) Glucagon
A) CagA B) VacA C) PicB D) OipA
A) An alkaline environment B) Lack of stomach acid C) Viral infection D) An acidic environment
A) TNF B) IL1B C) CagA D) BabA
A) The small intestine B) The gallbladder C) The esophagus D) The stomach
A) Chronic dehydration B) Helicobacter pylori infection C) Excessive intake of spicy foods D) Lack of physical exercise
A) Urea breath test B) Urine analysis C) Liver function test D) MRI scan
A) For all gastric ulcer cases B) When endoscopic treatment is unsuccessful C) Only in elderly patients D) As the first line of treatment
A) They kill H. pylori bacteria B) They neutralize stomach acid C) They help heal the stomach lining D) They can cause or worsen ulcers
A) Teenagers B) Older people C) Young adults D) Children
A) Heartburn B) Nausea C) Belching D) Waterbrash
A) COX-2 B) COX-1 C) Pepsinogen D) Lipoxygenase
A) Excessive spice consumption B) Physiological stress from serious health problems C) High caffeine intake D) Chronic psychological stress
A) Esophagogastroduodenoscopy (EGD) B) Blood antibody test C) Stool antigen test D) Barium contrast x-ray
A) Presence of H. pylori colonies B) Increased blood flow to the stomach C) Free gas underneath the diaphragm D) Enlargement of the stomach lining
A) Antibiotics B) Proton-pump inhibitors C) Antacids D) Anticoagulants
A) Barium sulfate B) Hydrochloric acid C) Nitrogen gas D) Radioactive carbon
A) Over age 30 B) Over age 45 C) Over age 60 D) Over age 18
A) Carcinoid B) Adenocarcinoma C) Lymphoma D) Gastrinoma
A) A false positive result B) A highly accurate result C) A false negative result D) Severe allergic reaction
A) Exactly 10 g/dL B) Greater than 7 g/dL C) Greater than 12 g/dL D) Less than 5 g/dL
A) MELD score B) Child-Pugh score C) APACHE II score D) Glasgow-Blatchford score
A) Antifibrinolytics B) Crystalloids C) Prokinetic agents D) Antacids
A) 80 mg bolus followed by 8 mg per hour B) 40 mg bolus followed by 4 mg per hour C) 100 mg bolus followed by 10 mg per hour D) 20 mg bolus followed by 2 mg per hour
A) 72 hours B) 48 hours C) 96 hours D) 24 hours
A) Cautery B) Antifibrinolytic agents C) Tranexamic acid D) Oral PPIs
A) 1997 B) 1984 C) 1958 D) 1982
A) Severe abdominal pain B) Nausea C) Halitosis D) Vomiting
A) Karolinska Medical Review B) New England Medical Journal C) Australian Medical Journal D) Journal of Molecular Biology
A) 1984 B) 1997 C) 2005 D) 1998
A) Atlanta, Georgia B) Stockholm, Sweden C) Athens, Greece D) UWA in Perth, Western Australia
A) Antibiotics B) A petri dish culture C) Spicy food extracts D) Mastic gum
A) High effectiveness B) Immediate elimination C) No effect D) Moderate improvement
A) Microbiologist B) Nobel Laureate C) Gastroenterologist D) General practitioner |